Just Plain Ruff

Ok, you give the weight appropriate dose of D25 twice and his sugar is now a nice 90. The heart rate is still 190+ and he is still presenting the way he did when you got him. What do we want to do now? this kid's still in big trouble - you note gasping breaths now.

no allergies no meds glucometer reading - Low No seizures I don't believe the sugar would produce a rate of 210 The mexican clinic ran a glucose check - it also was low, they put 10 spoonfuls of sugar in his pedialyte and told them to hoof it over to the US Nothing else done by the clinic- this was a one room mexican clinic with a overworked nurse and a overwhelmed doc.

true true true and the answer to one of the questions did say that radiographs for this case were not needed due to the presentation of the guy. If anyone wants more of these types of scenarios let me know.

There is no trauma noted nor is there any admission of trauma. To throw a wrench in the deal - the family took him to a Mexican medical clinic in Mexico and they told them to cross the border and call your ems system to come get them. The parents speak excellent english.

you are able (miraculously) get a scalp IV 3 lead monitor showing very narrow complex tachycardia, regular in rate 12 lead - you get it and it says SVT on it. (No picture available) Baby LOC Is responsive only to that iv stick. I would be pissy too if you stuck me in the head with a needle I'm thinking of one other quick test you can do to determine if there is a easy fix to the babies LOC(think no food or drink for 17 hours) Resp rate is now 60 Pulse rate 210 now Loc dropping - no response to pain ful stimuli

You get your tail on board. Vitals are as above Child is not looking good. You re-request helicopter and it's still transporting. You have a ambulance full of drugs and stuff. You also notice a red spot on his sclera of his right eye. His extremities are cool with thready pulses and a delayed capillary refill time of 5 to 6 seconds

I'll get one today for everyone

Wendy - bingo but let's go further. A 26-year-old man with an unknown past medical history arrives to the emergency department (ED) by ambulance. He had been driving his car while unrestrained and was involved in a high-speed motor vehicle collision. There was airbag deployment and significant front-end damage to the vehicle, with intrusion into the passenger compartment of the car. The patient was extricated from the vehicle and placed on a backboard, and a cervical collar was placed by EMS. A non-rebreather facemask and 1 peripheral intravenous (IV) line were placed in the field. On arrival to the hospital, the patient is ill-appearing and combative. His initial vital signs are a heart rate of 117 bpm, a blood pressure of 85/50 mm Hg, a respiratory rate of 32 breaths/min, and an oxygen saturation of 91% on the non-rebreather mask. On primary survey, his oropharynx is clear, his airway is patent, and his trachea appears to be shifted to the right of midline. On auscultation, the patient's breath sounds are decreased over the left chest. Percussion of the left chest demonstrates hyperresonance. His carotid pulse is weakly palpable, and his jugular venous pulse is elevated. The patient receives a Glasgow Coma Scale score of 12. The patient's clothing is removed, revealing no obvious deformities or areas of bleeding. The patient's abdomen is soft, without any tenderness to palpation. His pelvis is stable. Standard trauma x-rays, including an anteroposterior (AP) chest and pelvis scan, are performed after the primary survey. A complete secondary survey is postponed because of the patient's poor clinical condition. A second large-bore peripheral intravenous line is placed, and the patient begins to receive a bolus of 1000 cc of normal saline under pressure. A decision to perform an emergent procedure is made. Immediately after the procedure is performed, the patient is noted to have a dramatic clinical improvement. Subsequent to the procedure, the patient has a pulse of 105 bpm, a blood pressure of 95/60 mm Hg, a respiratory rate of 22 breaths/min, and an oxygen saturation of 98% on the non-rebreather mask. The secondary survey is completed, revealing no major injuries. Additionally, the chest radiograph (see Figure 1) confirms the suspected clinical diagnosis that prompted the emergent procedure.

that is a good one

Yes, good luck on your EMT class but please remember it's not rocket science. Go deeper in the items that are giving you trouble. You will find that EMT class is not as hard as you are making it out to be. For every 1 hour of class time spend at least 1 hour studying or better yet 4 hours. That's the number they use in college. If you need additional resources I can probably find them for you on the net so pm me and I'll try to help. If they are talkking about the heart you should learn about the cardiovascular system but don't go overboard and try to learn EKG's and such. That's extraneous crap you won't need until medic school oh yeah, make sure to have fun. And one other thing, post questions here often if you ahve them. Don't be afraid to pm those you trust with questions.

I want to add, a quality work life balance is KEY KEY KEY KEY KEY. Did I say that enough. If your life revolves around EMS (whackers I say whackers) then you will have no other hobbies or interests. I know a lot of people who would rather be at the station and "available" to run calls then you will get burned out To have a quality life out of work you need to have hobbies and interests that extend away from the firehouse/ems station. If that means you don't take your pager with you when you go out to eat or you don't answer your cell phone when you are on your days off then so be it. The ones who I see burn out fast are the ones who don't think that the county or EMS system can do without them. We all know those types of people right?

You are called to the very rural residence to a infant in distress. This is a 5 day old baby with rapid respirations and poor color. They called because the child has not been feeding and thier car is broken down so they have no way to get to the doctor. You have no air ambulance support due to a major disaster 100 miles north of you that required all the helicopters in your service area. You have a full complement of ALS equipment and there is a guarantee that you won't run out of any of your meds and your monitor is completely charged with 5 extra batteries. In other words, you are the paramedic, you have one EMT and that's it. No first responders except one deputy sheriff that is already there and is using blow by oxygen on the baby. What are your first steps.

Another scenario and educational piece from Medline CME by WebMD A 26-year-old man is involved in a high-speed motor vehicle collision. The patient’s oropharynx is clear, his airway is patent, and his trachea appears to be shifted to the right of midline. The patient’s breath sounds are decreased and percussion of the left chest demonstrates hyperresonance. Standard trauma x-rays, including an anteroposterior (AP) chest and pelvis scan, and an emergent procedure are performed. What is the underlying pathophysiology, and what procedure was performed?

Which of the following conditions is NOT part of the pathophysiologic process in a near-drowning or drowning injury? hypoxia with ischemia asphyxia oxygen toxicity Correct Answer irreversible tissue injury The pathophysiology in a near-drowning or drowning injury begins with initial immersion and asphyxia. This condition progresses to tissue hypoxia and ischemia that is initially reversible. If proper gas exchange via respiration is not restored, the injured tissues will suffer irreversible injury. Once irreversible tissue injury has occurred, the patient's outcome will depend upon the extent of the injury. Oxygen toxicity is the incorrect answer because this condition is related to an increased partial pressure of oxygen (PO2) within the body, whereas near-drowning and drowning are a result of decreased levels of oxygen (hypoxia). Boyle's Law is an important principle in physics that influences how the diving or hyperbaric environment affects our body. Which of the following variables is NOT related to Boyle's Law? temperature respiratory rate Correct Answer volume pressure Additional information on the answers: Which of the following conditions is NOT part of the pathophysiologic process in a near-drowning or drowning injury? Answer: oxygen toxicity The pathophysiology in a near-drowning or drowning injury begins with initial immersion and asphyxia. This condition progresses to tissue hypoxia and ischemia that is initially reversible. If proper gas exchange via respiration is not restored, the injured tissues will suffer irreversible injury. Once irreversible tissue injury has occurred, the patient's outcome will depend upon the extent of the injury. Oxygen toxicity is the incorrect answer because this condition is related to an increased partial pressure of oxygen (PO2) within the body, whereas near-drowning and drowning are a result of decreased levels of oxygen (hypoxia). Boyle's Law is an important principle in physics that influences how the diving or hyperbaric environment affects our body. Which of the following variables is NOT related to Boyle's Law? Boyle's Law states that at a constant temperature the volume of a gas varies inversely with its pressure. This law dictates the volume of gas required for a normal respiration at a given depth (pressure) and temperature. A person's respiratory rate will affect how quickly a given volume of gas is utilized at a given depth and temperature; however, the respiratory rate is not affected directly by depth (pressure).

You slacker, you should have posted it before me. here are the questions for the group from this reading Which of the following conditions is NOT part of the pathophysiologic process in a near-drowning or drowning injury? ____ hypoxia with ischemia ____ asphyxia ____ oxygen toxicity _____ irreversible tissue injury Boyle's Law is an important principle in physics that influences how the diving or hyperbaric environment affects our body. Which of the following variables is NOT related to Boyle's Law? ____ temperature ____ respiratory rate ____ volume ____ pressure

long hours bitchy nurses cantankerous docs stinky and crazy partners long hours oh did I already say long hours low pay low respect but if you work the job right there is tremendous satisfaction that you did make a difference in someone's life. Even if you held the hand of a little ole' lady to the ER after she fell and cut her head - and you comforted her then you made a difference This might be a sign of where you are doing your clinicals - it makes one think about whether you want to work for that particular service. If there are more than 20% of their force that you are seeing are complaining then there may be something deeper going on at that service to make their people burned out.

The correct answer was ---- Hypoxia caused by an improper gas mixture and here is how we got there::::: The link to the CT - found here and at the bottom of the post http://images.medscape.com/pi/editorial/ca...y14680.fig1.gif This incident was characterized as a near-drowning with aspiration of seawater secondary to hypoxia-induced unconsciousness caused by an improper gas mixture. The images from the chest CT scan revealed alveolar-type attenuations throughout the lungs, with focused attenuation in the left lower lobe. The mediastinum was normal and no evidence of pneumothorax was seen on the CT scan. A near-drowning is defined by Dorland's as "survival for any length of time after submersion in water and temporary suffocation; it sometimes ends with secondary drowning".[3] This incident was further categorized as a wet near-drowning because the patient aspirated seawater; this characterization is in contrast to a dry process, in which laryngospasm prevents the aspiration of water. The prognosis after near-drowning varies substantially. The 2 most influential factors are the duration of time the patient was submerged and the temperature of the water (cold water decreases cellular metabolism). These factors are ultimately related to the pathophysiology progressing from initial asphyxia, to hypoxia with ischemia and reversible tissue injury, to irreversible tissue injury with potential death from secondary drowning. Near-drowning is typically the result of an underlying event or condition that contributes to unintentional immersion or immersion with incapacitation, such as trauma, intoxication, seizure, hypothermia, dysrhythmia, shallow-water blackout, and, for divers, a lost or improper breathing source.[6] Auerbach emphasized the importance of determining if the near-drowning or drowning was wet (aspiration) or dry (no aspiration).[1] Although the mechanism of injury remains unchanged, aspiration of water in a near-drowning can affect respiration by washing out surfactant or injuring the alveolar membrane. In addition, aspiration increases the risk of a secondary pneumonia, which can further diminish respiration. One theoretical effect of wet drowning, which has been demonstrated in animal models and has caused some controversy, is electrolyte disturbance; however, this has not been convincingly observed in humans.[2] In this case, an improper breathing source resulted in hypoxia, which led to near-drowning. The diver was using a closed-circuit rebreathing apparatus, which adjusted the PO2 depending on his depth. PO2 is adjusted to avoid seizures caused by oxygen toxicity. The closed-circuit rebreathing system is essentially free of bubbles because the diver is inhaling and exhaling into a closed system. The closed system has a scrubber to remove carbon dioxide, an oxygen source, and a source of inert gas (eg, nitrogen). As a diver descends, increasing amounts of inert gas are introduced into the system, decreasing the amount of oxygen. This type of system is commonly used because it allows a diver to remain at depth for prolonged periods and because it reduces the amount of inert gas the tissues absorb. By comparison, the standard self-contained underwater breathing apparatus (SCUBA) used in most recreational dives is an open system, in which the exhaled breath is released into the water. Although a closed-circuit rebreathing system can be advantageous, mechanical failure and operator error are inherent risks. The most common mechanical failures are scrubber failure and delivery of an improper PO2 mixture. With scrubber failure, a diver can be incapacitated by hypercapnia. With an improper PO2 mixture, a diver can be incapacitated from either hypoxia or hyperoxia (hyperoxia can result in oxygen toxicity). Human error can occur when the PO2 is adjusted manually or when a diver is ascending or descending too quickly and does not let the apparatus adjust the pressures accurately. In the case of ascending too quickly, an event similar to shallow-water blackout occurs. In a shallow-water blackout, a person hyperventilates before breath-hold diving (diving without any external breathing apparatus) and metabolizes oxygen from the lungs while at depth. If the breath-hold diver metabolizes too much oxygen at depth, the PO2 is insufficient to sustain consciousness as he or she nears the surface, and blackout results. This event almost always ends in drowning, unless it is observed by others. The same mechanism can occur with a closed-circuit rebreathing apparatus if the PO2 is too low to sustain consciousness as the diver surfaces. In this case, it was clinically impossible to determine if the cause of the diver's hypoxia was mechanical failure or operator error. The diving apparatus must be thoroughly inspected to determine if a mechanical failure occurred. An arterial gas embolism (AGE) was clinically ruled out as the inciting event. The low PO2 during the ascent, the sensation of graying out, and the consistently normal neurologic findings after the incident were the key findings in this case. The patient's labored breathing, pink sputum, and diminished breath sounds on the left side suggested pulmonary barotrauma—specifically, pneumothorax. Given the clinical picture, hyperbaric oxygen therapy was not indicated and was thought to be potentially harmful because of the possibility of pneumothorax. The 2 most important principles of physics in regard to diving are the Boyle law and the Henry law. The Boyle law states that, at a constant temperature, the volume of a gas varies inversely with its pressure. The Henry law states that the amount of gas dissolved in liquid or in human tissues at a given temperature is a function of its partial pressure. The Boyle law and the Henry law are the underlying mechanisms responsible for the 2 major types of hyperbaric injury: pulmonary barotrauma and decompression sickness (DCS), respectively. Also called pulmonary overinflation syndrome, pulmonary barotrauma occurs when gas expansion in the lungs ruptures the lung tissue. Typically, this is caused by breath-holding on ascent, by a rapid or unconscious ascent, or by air trapped in the lungs on ascent. Air trapping is most commonly caused by asthma, secretions, pneumonia, and blebs. AGE, pneumothorax, mediastinal emphysema, subcutaneous emphysema, and pneumopericardium can result. An AGE is the result of a lung rupture that also ruptures veins or capillaries, shunting gas to the left side of the heart and distributing it to end organs. The most common signs and symptoms are cranial nerve deficits, paralysis or weakness, sensory abnormalities, poor coordination, and unconsciousness. More than 98% of AGEs manifest within 10 minutes after the initial injury. Pneumothorax or tension pneumothorax can result if gas is released into the pleural space. Pneumopericardium, mediastinal emphysema, and subcutaneous emphysema occur when gas escapes through the connective tissues in the thorax. DCS is also known as decompression illness, the bends, and Caisson disease. DCS is caused by the formation of microbubbles in the tissues as a result of decreasing ambient pressure. Its pathophysiology is based on the mechanical and nonmechanical effects of the bubbles. The mechanical effects are limited to hypoxic injury caused by blocked blood flow and compression of localized tissues and surrounding structures. The nonmechanical effects are diverse; however, those most widely studied and understood are an inflammatory response, an intravascular aggregation of platelets and leukocytes, and stimulation of the clotting cascade. The mechanical and nonmechanical effects combine to create the clinical picture of DCS. Most cases become apparent within the first hour, and >98% manifest within 24 hours. DCS is clinically separated into 2 types: type I and type II. DCS type I causes musculoskeletal joint pain, skin itching and marbling, and lymphatic swelling or tender lymph nodes. DCS type II leads to pulmonary, vestibular, or neurologic signs and symptoms. Type I is by far more common than type II, and the prognosis is typically more favorable for type I than for type II. Treatment is the same for type I and type II. Hyperbaric oxygenation treats both the mechanical and nonmechanical effects, by crushing the bubbles in the blood or tissues and by increasing the oxygen levels in the injured tissue. The diver in this case was admitted to the hospital for aspiration pneumonitis after the near-drowning episode. He was observed for a period of 48 hours. There were no episodes of hypoxia, and no deterioration of his clinical condition was noted during this period. He was discharged to home in good health. I will post the CME questions in a subsequent post. Hey Doc, this is the only image given in the scenario - http://images.medscape.com/pi/editorial/ca...y14680.fig1.gif

Don't bet on it. Democratic chairpersons don't get the same consideration as let's say, major league ball players, NBA players, NFL players, mayors and city councilman.

you are only just starting and you have some really good stuff. I'll mark you on my fav's and read up. It's nice to see someone on the other side of the water blogging about their service. Thanks for the early morning read.

I will refrain from posting anything till some more people answer. The answer is really simple if you look at it. The answer is similar to why you get light headed when you hold your breath.

Thanks to Medline CME for this scenario A Diver Unconscious at the Surface After a Recreational Dive Background A 22-year-old male recreational diver was performing an open-ocean dive using a closed-circuit rebreathing apparatus with a group of fellow recreational divers. When he surfaced, he was unconscious; observers at the scene do not recall if his mouthpiece was in place. He was immediately placed into a nearby dive boat and taken to the emergency department (ED). On-site divers trained in basic life support noted that the diver was initially unconscious and apneic, though he did have a pulse. They delivered 2 rescue breaths, after which the diver began breathing spontaneously. He quickly regained consciousness and was immediately given oxygen 10 L/min by face mask. Within about 30 seconds, he began coughing pink, frothy sputum. The diver reported feeling normal except for fullness in his chest and difficulty in taking a deep breath. Focused neurologic examinations and assessments of his vital signs were performed at 5-minute intervals. The findings were reported as normal and stable during the trip to shore, which was approximately 30 minutes. On presentation at the ED, the patient states that he had difficulty maintaining a proper partial pressure of oxygen (PO2) throughout his dive. He believes that his PO2 was too low and that his vision was graying out as he ascended to the surface. According to the dive master, the group's diving profile was consistent with a no-decompression dive, with a maximum depth of 75 feet of seawater. The other divers are uncertain about how long the patient was unconscious, though they remember seeing him conscious at approximately 10 feet of seawater during the ascent. If correct, this means that the patient was unconscious for about 20 seconds underwater at a standard ascent rate of 30 ft/min. On physical examination, the patient's face-mask reservoir contains approximately 20 mL of pink, frothy sputum. The patient is a healthy-appearing man in no acute distress, but his work of breathing is increased. He has a pulse of 80 bpm, a blood pressure of 104/89 mm Hg, and an oxygen saturation of 98% while breathing oxygen 10 L/min by face mask and 92% while breathing room air. The neurologic examination reveals no deficits. His speech is normal, and there is no fullness of the neck, jugular venous distention, or tracheal deviation. Auscultation of the left lower lung field reveals diminished breath sounds. The remainder of the physical examination yields no abnormalities. A chest radiograph is unrevealing. A computed tomography (CT) scan of his chest is obtained (see Figure 1). Questions: What was the cause of the loss of consciousness during the diver's ascent? Cerebral gas embolism __ Oxygen toxicity caused by an improper gas mixture __ Scrubber failure leading to hypercapnia __ Hypoxia caused by an improper gas mixture __ I will post a follow up with lots more information from medline CME in the posts to come.

what a wonderful testimony to further education. A lot of us on this site and beyond can learn from these words by our esteemed colleague nremt-basic. Rock on dude.

Get me a ticket to an airoplane or whatever the title of that song is.

I did it again didnt I

haven't we discussed this in the past. I remember a distinct thread on it many months ago.