Arizonaffcep

And he's a hell of a nice guy. He will always stop to talk/discuss questions/etc (provided the timing is correct and he's not busy with a Pt). First time I met him when he started at UMC in Tucson, he introduced himself as "Peter." How many well known trauma surgons do that? He's great! Out of curiosity, I know this thread is a few months from the last post, but I just wrote a paper on permissive hypotension for Lifeline, and was wondering if anyone had any interest in reading it. It deals with a bunch of different aspects all involved with blood pressure and trauma care. Including cited references to the thought that a systolic of 80mmHg will "pop clots"...

Well hell, I can see that on myself and I'm about 20 lbs overweight . That being said, it's the pressure wave is what you are seeing affecting the surrounding tissues, and NOT the actual outline of the aorta itself. That'd be really impressive. Maybe in Ethiopia?

As I was reading the posts for this, I don't remember seeing anyone say the science behind the "new" ACLS changes (they are also coming out with new guidelines this year as well folks), for the AHA. It was the largest study done, certainly by the AHA (possibly ever) and encompassed medicine from literally all over the world. What was discovered was that because the coronary arteries are "filled" on the "back pressure" from the aorta, it does take a significant amount of time and energy (in this case energy=compressions) to bring the perfusion of the myocardium to a state where the muscle is more "accepting" of an electrical charge. Another reason that they are saying "don't stop compressions for LONGER than 10 SECONDS" is because they found that the cardiac output during compressions is at BEST 1/3 of a normal contraction, and the assumption was that it was higher than that prior to the release of the studies. That being said, there really is no reason that you should stop compressions for longer than 10 seconds, especially with the availability of alternative airways. I would prefer the combi-tube or something similar instead of an LMA, but the reality is either will work. As an addendum to the AHA studies, they found that for every 10 second stop (2 minutes or 5 cycles of 30 compressions and 1 breath-BVM, non-secured airway) it takes about 15-20 compressions to bring that perfusion level in the myocardium back up to the point where it was before the 10 second stop. So, in essence, even with only a 10 second stop, at least 1/2 of the compressions of the next "round" are just used to bring the perfusion back up and not a maintenance.

What's his 12 lead look like?

Good call!

Sorry for the development of a typing lisp...not sure what happened. In reading over this again, would probably go with a breathing treatment as well.

CH, from the research I've done in the past year (first partner on the helicopter was really into this etomidate stuff) it's not a single does that does the bad, as levels will return to normal and if you look at the output for 24 hours, there is little difference between those that didn't get it and those that got a single dose. However, the continued use (ie etomidate drip) is what all the commotion is about. I would quote the studies, but taking a break from chart writing and don't have the time to re-research them.

My company doesn't have any policy for kidnapped crew...but don't think that'd be even a small potential (possible, just extrodiarily improbable), as we are a crew of 3 (pilot, medic, RN) and either respond to a scene that's full of personnel already along with PD-typically to shut down a road for landing-or to a facility (rural hospital/clinic)-so the potential is VERY low. But...we do have a PAIP (post accident/incident plan) in place for missing aircraft. The last drill we did, our dispatch took 7 minutes from when we had an unscheduled landing to our pagers going off that they had our GPS location, had notified the program manager and initiated the PAIP. Seems like a good time frame to me, as in talking with other flight crews, they've done the same and sat for over 45 minutes... See question above. Yes, while we don't have handhelds, our aircraft are equipted with the lattest radio/gps safety stuff. Infact, it's the newest dual band thingy.

From the founds of it,I would think infection moving along to sepsis. Pneumonia? For me, continue any meds that need to be continued (ie abx), other than that, supportive measures. Give him as much O2 as they can tolerate, possibly fluid challenge for decreased B/P. Reason for possible fluid challenge with wet lungs-infection causing wet lungs vs CHF/ARDS, etc. In my eyes, this seems to be a fairly obvious infection-hence the temp.

Quite the clever marketing scheme...

How thin was this patient? Seriously...on an "regular sized" adult, you shouldn't be able to see that. I just did a paper on permissive hypotension, and came across a quote (although for the life of me I can't find it again). However, it did say something along the line of a patient can bleed in the realm of 2L into the abdomen without it being noticed visually (distending). This being said...I would find the whole sitution implausable.

Ok...so I haven't posted on here in like...a year or more shame on me. Do damned busy. Only words of wisdom I might have regarding fluids bolus with suspected CVA is an OBVIOUS Cushings Triad, where their MAP would need to be adjusted. But...that's rare and WAY late in the bleed. Only time I can think of.