Epi-Respirations?

[fiznat]

While I'm reading, care to offer a synoposis on your understanding of the pathophysiology in question?

[AZCEP]

The brain is failing so the chemoreceptor center is maintaining function at a "pseudo-elevated" rate based on the amount of CO2. If the brain would regain perfusion the respiratory rate would increase accordingly, but because it can't the only drive is from the brain stem, thus the rate that is present.

[FL_Medic]

The brain is failing so the chemoreceptor center is maintaining function at a "pseudo-elevated" rate based on the amount of CO2. If the brain would regain perfusion the respiratory rate would increase accordingly, but because it can't the only drive is from the brain stem, thus the rate that is present.

That makes sense to me, thanks

[Ace844]

The brain is failing so the chemoreceptor center is maintaining function at a "pseudo-elevated" rate based on the amount of CO2. If the brain would regain perfusion the respiratory rate would increase accordingly, but because it can't the only drive is from the brain stem, thus the rate that is present.

Well put, I'll also add the possibility that if the CPR was effective or a mechanical device used the brain could have recieved soem perfusion or had it restored either with ROSC, or augmented by the meds enough to cause the pneumotactic center to respond as mentioned previously to the high Co2 levels and acidotic state which would be present post resus.

ACE

[FL_Medic]

Well put, I'll also add the possibility that if the CPR was effective or a mechanical device used the brain could have recieved soem perfusion or had it restored either with ROSC, or augmented by the meds enough to cause the pneumotactic center to respond as mentioned previously to the high Co2 levels and acidotic state which would be present post resus.

ACE

The CPR was phenominal

[Ace844]

there ya go then..that seems to be the answers you were looking for.

ACE